[1] Defronzo RA, Tripathy D. Skeletal muscle insulin resistance is the primary defect in type 2 diabetes [J]. Diabetes Care, 2009, 32(Suppl 2): S157-S163.
[2] Tam CS, Powe JE, Markovic TP, et al. The effects of high-fat feeding on physical function and skeletal muscle extracellular matrix [J]. Nutr Diabetes, 2015, 5: e187.
[3] Kang L, Lantier L, Kennedy A, et al. Hyaluronan accumulates with high-fat feeding and contributes to insulin resistance [J]. Diabetes, 2013, 62(6): 1888-1896.
[4] Kang L, Mayes WH, James FD, et al. Matrix metalloproteinase 9 opposes diet-induced muscle insulin resistance in mice [J]. Diabetologia, 2014, 57(3): 603-613.
[5] Kang L, Ayala JE, Lee-Young RS, et al. Diet-induced muscle insulin resistance is associated with extracellular matrix remodeling and interaction with integrin alpha2beta1 in mice [J]. Diabetes, 2011, 60(2): 416-426.
[6] Scheede-Bergdahl C, Bergdahl A, Schjerling P, et al. Exercise-induced regulation of matrix metalloproteinases in the skeletal muscle of subjects with type 2 diabetes [J]. Diab Vasc Dis Res, 2014, 11(5): 324-334.
[7] Gillies AR, Lieber RL. Structure and function of the skeletal muscle extracellular matrix [J]. Muscle Nerve, 2011, 44(3): 318-331.
[8] Grounds MD, Sorokin L, White J. Strength at the extracellular matrix-muscle interface [J]. Scand J Med Sci Sports, 2005, 15(6): 381-391.
[9] Berria R, Wang L, Richardson DK, et al. Increased collagen content in insulin-resistant skeletal muscle [J]. Am J Physiol Endocrinol Metab, 2006, 290(3): E560-565.
[10] Sishi B, Loos B, Ellis B, et al. Diet-induced obesity alters signalling pathways and induces atrophy and apoptosis in skeletal muscle in a prediabetic rat model [J]. Exp Physiol, 2011, 96(2): 179-193.
[11] Pincu Y, Linden MA, Zou K, et al. The effects of high fat diet and moderate exercise on TGFbeta1 and collagen deposition in mouse skeletal muscle [J]. Cytokine, 2015, 73(1): 23-29.
[12] Watts R, Mcainch AJ, Dixon JB, et al. Increased Smad signaling and reduced MRF expression in skeletal muscle from obese subjects [J]. Obesity (Silver Spring), 2013, 21(3): 525-528.
[13] Ge X, Vajjala A, Mcfarlane C, et al. Lack of Smad3 signaling leads to impaired skeletal muscle regeneration [J]. Am J Physiol Endocrinol Metab, 2012, 303(1): E90-102.
[14] Williams AS, Kang L, Wasserman DH. The extracellular matrix and insulin resistance [J]. Trends Endocrinol Metab, 2015, 26(7): 357-366.
[15] Solomon TP, Haus JM, Li Y, et al. Progressive hyperglycemia across the glucose tolerance continuum in older obese adults is related to skeletal muscle capillarization and nitric oxide bioavailability [J]. J Clin Endocrinol Metab, 2011, 96(5): 1377-1384.
[16] Kodama K, Horikoshi M, Toda K, et al. Expression-based genome-wide association study links the receptor CD44 in adipose tissue with type 2 diabetes [J]. Proc Natl Acad Sci U S A, 2012, 109(18): 7049-7054.
[17] Fogelstrand P, Boren J. Treatment of hyaluronan accumulation ameliorates high-fat diet-induced insulin resistance in mice [J]. Diabetes, 2013, 62(6): 1816-1817.
[18] Odegaard JI, Chawla A. Pleiotropic actions of insulin resistance and inflammation in metabolichomeostasis [J]. Science, 2013, 339(6116): 172-177.
[19] Zong H, Bastie CC, Xu J, et al. Insulin resistance in striated muscle-specific integrin receptor beta1-deficient mice [J]. J Biol Chem, 2009, 284(7): 4679-4688.
[20] Kang L, Mokshagundam S, Reuter B, et al. Integrin-linked kinase in muscle is necessary for the development of insulin resistance in diet-induced obese mice [J]. Diabetes, 2016, 65(6): 1590-1600.
[21] Mcdonald PC, Fielding AB, Dedhar S. Integrin-linked kinase－essential roles in physiology and cancer biology [J]. J Cell Sci, 2008, 121(Pt 19): 3121-3132.
[22] Gupta A, Bisht B, Dey CS. Focal adhesion kinase negatively regulates neuronal insulin resistance [J]. Biochim Biophys Acta, 2012, 1822(6): 1030-1037.
[23] Viglino C, Montessuit C. A role for focal adhesion kinase in the stimulation of glucose transport in cardiomyocytes [J]. J Cell Biochem, 2017, 118(4): 670-677.
[24] Lin D, Chun TH, Kang L. Adipose extracellular matrix remodelling in obesity and insulin resistance [J]. Biochem Pharmacol, 2016, 119: 8-16.
[25] Rodrigues SF, Tran ED, Fortes ZB, et al. Matrix metalloproteinases cleave the beta(2)-adrenergic receptor in spontaneously hypertensive rats [J]. Am J Physiol Heart Circ Physiol, 2010, 299(1): H25-H35.
[26] Delano FA, Schmid-Schonbein GW. Proteinase activity and receptor cleavage: mechanism for insulin resistance in the spontaneously hypertensive rat [J]. Hypertension, 2008, 52(2): 415-423.
[27] Tinahones FJ, Coin-Araguez L, Mayas MD, et al. Obesity-associated insulin resistance is correlated to adipose tissue vascular endothelial growth factors and metalloproteinase levels [J]. BMC Physiol, 2012, 12: 4.
[28] Hollborn M, Stathopoulos C, Steffen A, et al. Positive feedback regulation between MMP-9 and VEGF in human RPE cells [J]. Invest Ophthalmol Vis Sci, 2007, 48(9): 4360-4367.
[29] Tooke JE, Goh KL. Endotheliopathy precedes type 2 diabetes [J]. Diabetes Care, 1998, 21(12): 2047-2049.
[30] Rullman E, Norrbom J, Stromberg A, et al. Endurance exercise activates matrix metalloproteinases in human skeletal muscle [J]. J Appl Physiol (1985), 2009, 106(3): 804-812.
[31] Davis ME, Gumucio JP, Sugg KB, et al. MMP inhibition as a potential method to augment the healing of skeletal muscle and tendon extracellular matrix [J]. J Appl Physiol (1985), 2013, 115(6): 884-891.
[32] Hopps E, Lo Presti R, Montana M, et al. Gelatinases and their tissue inhibitors in a group of subjects with metabolic syndrome [J]. J Investig Med, 2013, 61(6): 978-983.
[33] Jaworski DM, Sideleva O, Stradecki HM, et al. Sexually dimorphic diet-induced insulin resistance in obese tissue inhibitor of metalloproteinase-2 (TIMP-2)-deficient mice [J]. Endocrinology, 2011, 152(4): 1300-1313.
[34] Kandalam V, Basu R, Abraham T, et al. TIMP2 deficiency accelerates adverse post-myocardial infarction remodeling because of enhanced MT1-MMP activity despite lack of MMP2 activation [J]. Circ Res, 2010, 106(4): 796-808.
[35] Menghini R, Menini S, Amoruso R, et al. Tissue inhibitor of metalloproteinase 3 deficiency causes hepatic steatosis and adipose tissue inflammation in mice [J]. Gastroenterology, 2009, 136(2): 663-672.e4.
