专题康复2030:国际康复政策与发展

基于ICF构建国家功能、残疾和健康的信息架构和数据体系

  • 李沁燚 ,
  • 邱卓英 ,
  • 陈迪 ,
  • 李欣 ,
  • 杨剑
展开
  • 1.中国智力和发展性残疾分级、评估和康复(运动)重点实验室,郑州大学,河南郑州市450001;
    2.中国康复研究中心康复信息研究所,世界卫生组织国际分类家族中国合作中心,北京市100068;
    3.郑州大学体育学院(校本部),河南郑州市450001;
    4.华东师范大学体育与健康学院,青少年健康评价与运动干预教育部重点实验室,上海市200241。
李沁燚(1986-),女,汉族,河南信阳市人,硕士,助理研究员,主要研究方向:ICF、残疾康复信息。

收稿日期: 2016-08-11

  网络出版日期: 2017-05-17

基金资助

国家自然科学基金项目(No.30772299)。

Construction of National Framework and Data Systems of Functioning, Disability, and Health of Information Using ICF 

  • LI Qin-yi ,
  • QIU Zhuo-ying ,
  • CHEN Di ,
  • LI Xin ,
  • YANG Jian
Expand
  • 1. China Key Laboratory of Classification, Evaluation and Rehabilitation (Sport) of Intellectual and Development Disability, Zhengzhou University, Zhengzhou, Henan 450001, China;
    2. Research Institute of Rehabilitation Information, China Rehabilitation Research Center, WHO-FIC Collaborating Center in China, Beijing 100068, China;
    3. School of Sport Science, Zhengzhou University (Main Campus), Zhengzhou, Henan 450001, China;
    4. Key Laboratory of Health Evaluation and Sport Intervention of Ministry of Education, School of Sport and Health, East China Normal University, Shanghai 200241, China

Received date: 2016-08-11

  Online published: 2017-05-17

摘要

运用ICF有关功能、残疾和健康的理论与分类工具,通过对功能、残疾和健康数据进行分析,基于ICF对功能和残疾数据进行标识、度量和测度分析,构建国家功能、残疾和健康的信息架构和数据体系。实现国际和国家间功能、残疾和健康数据信息之间的比较,用于监测康复服务质量和残疾人生活质量与发展状况,实施世界卫生组织康复指南:《健康服务体系中的康复(2017)》。 

本文引用格式

李沁燚 , 邱卓英 , 陈迪 , 李欣 , 杨剑 . 基于ICF构建国家功能、残疾和健康的信息架构和数据体系[J]. 中国康复理论与实践, 2017 , 23(4) : 385 -389 . DOI: 10.3969/j.issn.1006-9771.2017.04.004

Abstract

Identification, measure and metrics of functioning, disability and health data has been developed using ICF approach to construct national information framework and health data systems of functioning, disability and health. This framework and system make countries' data comparable. It is useful to monitor the quality of rehabilitation services and develop services for people with disabilities as recommended by World Health Organization Guideline: Rehabilitation in Health Systems.

参考文献

[1] Davidoff GN, Roth EJ, Richards JS. Cognitive deficits in spinal cord injury: epidemiology and outcome [J]. Arch Phys Med Rehabil, 1992, 73(3): 275-284.
[2] Davidoff G, Morris J, Roth E, et al. Cognitive dysfunction and mild closed head injury in traumatic spinal cord injury [J]. Arch Phys Med Rehabil, 1985, 66(8): 489-491.
[3] Wilmot CB, Cope DN, Hall KM, et al. Occult head injury: its incidence in spinal cord injury [J]. Arch Phys Med Rehabil, 1985, 66(4): 227-231.
[4] Richards JS, Brown L, Hagglund K, et al. Spinal cord injury and concomitant traumatic brain injury. Results of a longitudinal investigation [J]. Am J Phys Med Rehabil, 1988, 67(5): 211-216.
[5] Roth E, Davidoff G, Thomas P, et al. A controlled study of neuropsychological deficits in acute spinal cord injury patients [J]. Paraplegia, 1989, 27(6): 480-489.
[6] Lazzaro I, Tran Y, Wijesuriya N, et al. Central correlates of impaired information processing in people with spinal cord injury [J]. J Clin Neurophysiol, 2013, 30(1): 59-65.
[7] Di Russo F, Bultrini A, Brunelli S, et al. Benefits of sports participation for executive function in disabled athletes [J]. J Neurotrauma, 2010, 27(12): 2309-2319.
[8] Gordon E. Neuroimaging in neuropsychiatry [J]. Semin Clin Neuropsychiatry, 2002, 7(1): 42-53.
[9] Noriega DB, Savelkoul HF. Immune dysregulation in autism spectrum disorder [J]. Eur J Pediatr, 2014, 173(1): 33-43.
[10] Rivat C, Becker C, Blugeot A, et al. Chronic stress induces transient spinal neuroinflammation, triggering sensory hypersensitivity and long-lasting anxiety-induced hyperalgesia [J]. Pain, 2010, 150(2): 358-368.
[11] Widerstr?m-Noga EG, Felipe-Cuervo E, Broton JG, et al. Perceived difficulty in dealing with consequences of spinal cord injury [J]. Arch Phys Med Rehabil, 1999, 80(5): 580-586.
[12] Murray RF, Asghari A, Egorov DD, et al. Impact of spinal cord injury on self-perceived pre- and postmorbid cognitive, emotional and physical functioning [J]. Spinal Cord, 2007, 45(6): 429-436.
[13] Hess DW, Marwitz JH, Kreutzer JS. Neuropsychological impairments after spinal cord injury: a comparative study with mild traumatic brain injury [J]. Rehabil Psychol, 2003, 48(3): 151-156.
[14] Davidoff G, Morris J, Roth E, et al. Closed head injury in spinal cord injured patients: retrospective study of loss of consciousness and post-traumatic amnesia [J]. Arch Phys Med Rehabil, 1985, 66(1): 41-43.
[15] Davidoff G, Thomas P, Johnson M, et al. Closed head injury in acute traumatic spinal cord injury: incidence and risk factors [J]. Arch Phys Med Rehabil, 1988, 69(10): 869-872.
[16] Hovda DA. The neurophysiology of concussion [J]. Prog Neurol Surg, 2014, 28: 28-37.
[17] Wang Q, Wang Z, Zhu P, et al. Alterations of myelin basic protein and ultrastructure in the limbic system at the early stage of trauma-related stress disorder in dogs [J]. J Trauma, 2004, 56(3): 604-610.
[18] Zhang B, Huang Y, Su Z, et al. Neurological, functional, and biomechanical characteristics after high-velocity behind armor blunt trauma of the spine [J]. J Trauma, 2011, 71(6): 1680-1688.
[19] 冯东亮,南伟,伍亚民,等. 脊髓损伤大鼠的学习记忆功能与海马病理改变[J]. 中国康复理论与实践, 2015, 21(11): 1267-1272.
[20] Freund P, Weiskopf N, Ashburner J, et al. MRI investigation of the sensorimotor cortex and the corticospinal tract after acute spinal cord injury: a prospective longitudinal study [J]. Lancet Neurol, 2013, 12(9): 873-881.
[21] 侯景明. 脊髓损伤后脑结构和功能改变的多模态磁共振研究[D]. 重庆:第三军医大学, 2015.
[22] Nicotra A, Critchley HD, Mathias CJ, et al. Emotional and autonomic consequences of spinal cord injury explored using functional brain imaging [J]. Brain, 2006, 129(Pt 3): 718-728.
[23] Hilton BJ, Anenberg E, Harrison TC, et al. Re-establishment of cortical motor output maps and spontaneous functional recovery via spared dorsolaterally projecting corticospinal neurons after dorsal column spinal cord injury in adult mice [J]. J Neurosci, 2016, 36(14): 4080-4092.
[24] Han Q, Cao C, Ding Y, et al. Plasticity of motor network and function in the absence of corticospinal projection [J]. Exp Neurol, 2015, 267: 194-208.
[25] Hains BC, Black JA, Waxman SG. Primary cortical motor neurons undergo apoptosis after axotomizing spinal cord injury [J]. J Comp Neurol, 2003, 462(3): 328-341.
[26] Fumagalli F, Madaschi L, Caffino L, et al. Acute spinal cord injury reduces brain derived neurotrohic factor expression in rat hippocampus [J]. Neuroscience, 2009, 159(3): 936-939.
[27] Chang CM, Lee MH, Wang TC, et al. Brain protection by methylprednisolone in rats with spinal cord injury [J]. Neuroreport, 2009, 20(10): 968-972.
[28] Wu J, Stoica BA, Luo T, et al. Isolated spinal cord contusion in rats induces chronic brain neuroinflammation, neurodegeneration, and cognitive impairment. Involvement of cell cycle activation [J]. Cell Cycle, 2014, 13(15): 2446-2458.
[29] Wu J, Raver C, Piao C, et al. Cell cycle activation contributes to increased neuronal activity in the posterior thalamic nucleus and associated chronic hyperesthesia after rat spinal cord contusion [J]. Neurotherapeutics, 2013, 10(3): 520-538.
[30] Guerrero AR, Uchida K, Nakajima H, et al. Blockade of interleukin-6 signaling inhibits the classic pathway and promotes an alternative pathway of macrophage activation after spinal cord injury in mice [J]. J Neuroinflammation, 2012, 9: 40.
[31] Kipnis J, Gadani S, Derecki NC. Pro-cognitive properties of T cells [J]. Nat Rev Immunol, 2012, 12(9): 663-669.
[32] Skaper SD, Facci L, Giusti P. Neuroinflammation, microglia and mast cells in the pathophysiology of neurocognitive disorders: a review [J]. CNS Neurol Disord Drug Targets, 2014, 13(10): 1654-1666.
[33] Hotamisligil GS. Endoplasmic reticulum stress and the inflammatory basis of metabolic disease [J]. Cell, 2010, 140(6): 900-917.
[34] Wu J, Zhao Z, Kumar A, et al. Endoplasmic reticulum stress and disrupted neurogenesis in the brain are associated with cognitive impairment and depressive-like behavior after spinal cord injury [J]. J Neurotrauma, 2016 33(21): 1919-1935.
[35] Claydon VE, Krassioukov AV. Orthostatic hypotension and autonomic pathways after spinal cord injury [J]. J Neurotrauma, 2006, 23(12): 1713-1725.
[36] Phillips AA, Warburton DE, Ainslie PN, et al. Regional neurovascular coupling and cognitive performance in those with low blood pressure secondary to high-level spinal cord injury: improved by alpha-1 agonist midodrine hydrochloride [J]. J Cereb Blood Flow Metab, 2014, 34(5): 794-801.
文章导航

/

〈 〉