[1] van der Scheer JW, Hutchinson MJ, Paulson T, et al. Reliability and validity of subjective measures of aerobic intensity in adults with spinal cord injury: a systematic review[J]. PM R, 2018, 10(2): 194-207.
[2] Dietz V, Fouad K.Restoration of sensorimotor functions after spinal cord injury[J]. Brain, 2014, 137(Pt 3): 654-667.
[3] Saulino M.Spinal cord injury pain[J]. Phys Med Rehabil Clin N Am, 2014, 25(2): 397-410.
[4] Sohn HM, Hwang JY, Ryu JH, et al.Simvastatin protects ischemic spinal cord injury?from cell death and cytotoxicity through decreasing oxidative stress: in vitro primary cultured rat spinal cord model under oxygen and glucose deprivation-reoxygenation conditions[J]. J Orthop Surg Res, 2017, 12(1): 36.
[5] Ur K, Demiroz S, Bengu AS, et al.Serum endocan level and the severity of spinal cord injury[J]. Bratisl Lek Listy, 2018, 119(5): 298-301.
[6] Fehlings MG, Vaccaro A, Wilson JR, et al.Early versus delayed decompression for traumatic cervical spinal cord injury: results of the Surgical Timing in Acute Spinal Cord Injury Study (STASCIS)[J]. PLoS One, 2012,7(2): e32037.
[7] Nicholson JK, Connelly J, Lindon JC, et al.Metabonomics: a platform for studying drug toxicity and gene function[J]. Nat Rev Drug Discov, 2002, 1(2): 153-161.
[8] Jacob M, Malkawi A, Albast N, et al.A targeted metabolomics approach for clinical diagnosis of inborn errors of metabolism[J]. Anal Chim Acta, 2018, 1025: 141-153.
[9] Euceda LR, Andersen MK, Tessem MB, et al.NMR-based prostate cancer?metabolomics[J]. Methods Mol Biol, 2018, 1786: 237-257.
[10] Bunge RP, Puckett WR, Hiester ED.Observations on the pathology of several types of human spinal cord injury, with emphasis on the astrocyte response to penetrating injuries[J]. Adv Neurol, 1997, 72: 305-315.
[11] Hagen EM, Rekand T, Gilhus NE, et al.Traumatic spinal cord injuries–incidence, mechanisms and course[J]. Tidsskr Nor Laegeforen, 2012, 132(7): 831-837.
[12] Trivedi A, Olivas AD, Noble-Haeusslein LJ.Inflammation and spinal cord injury: infiltrating leukocytes as determinants of injury and repair processes[J]. Clin Neurosci Res, 2006, 6(5): 283-292.
[13] Siddiqui AM, Khazaei M, Fehlings MG.Translating mechanisms of neuroprotection, regeneration, and repair to treatment of spinal cord injury[J]. Prog Brain Res, 2015, 218: 15-54.
[14] Biglari B, Swing T, Child C, et al.A pilot study on temporal changes in IL-1β and TNF-α serum levels after spinal cord injury: the serum level of TNF-α in acute SCI patients as a possible marker for neurological remission[J]. Spinal Cord, 2015, 53(7): 510-514.
[15] Gensel JC, Zhang B.Macrophage activation and its role in repair and pathology after?spinal cord injury[J]. Brain Res, 2015, 1619: 1-11.
[16] Gaudet AD, Popovich PG.Extracellular matrix regulation of?inflammation in the healthy and injured spinal cord[J]. Exp Neurol, 2014, 258: 24-34.
[17] Mazzon E, Bruscoli S, Galuppo M, et al.Glucocorticoid-induced leucine zipper (GILZ) controls?inflammation?and tissue damage after?spinal cord injury[J]. CNS Neurosci Ther, 2014, 20(11): 973-981.
[18] Hulme CH, Brown SJ, Fuller HR, et al.The developing landscape of diagnostic and prognostic biomarkers for spinal cord injury in cerebrospinal fluid and blood[J]. Spinal Cord, 2017, 55(2): 114-125.
[19] Lukovic D, Stojkovic M, Moreno-Manzano V, et al.Concise review: reactive astrocytes and stem?cells?in?spinal cord injury: good guys or bad guys?[J]. Stem Cells, 2015, 33(4): 1036-1041.
[20] Ruschel J, Hellal F, Flynn KC, et al.Axonal regeneration. Systemic administration of epothilone B promotes axon regeneration after?spinal cord injury[J]. Science, 2015, 348(6232): 347-352.
[21] Hirokawa T, Zou Y, Kurihara Y, et al.Regulation of axonal regeneration by the level of function of the endogenous Nogo receptor antagonist LOTUS[J]. Sci Rep, 2017, 7(1): 12119.
[22] Dyck S, Kataria H, Alizadeh A, et al.Perturbing chondroitin sulfate proteoglycan signaling through LAR and PTPσ receptors promotes a beneficial inflammatory response following?spinal cord injury[J]. J Neuroinflammation, 2018, 15(1): 90.
[23] Dou F, Huang L, Yu P, et al.Temporospatial expression and cellular localization of oligodendrocyte myelin glycoprotein (OMgp) after traumatic spinal cord injury in adult rats[J]. J Neurotrauma, 2009, 26(12): 2299-2311.
[24] 坊爱红,张婷,孙丽,等. 脊髓损伤治疗的研究进展[J]. 中国综合临床, 2018, 34(1): 75-78.
[25] 杜盛超,张玮,胡浩然,等. 低温在脊髓损伤治疗中应用的研究进展[J]. 现代生物医学进展, 2017, 17(11): 2194-2196, 2177.
[26] Koda M, Nishio Y, Kamada T, et al.Granulocyte colony-stimulating factor (G-CSF) mobilizes bone marrow-derived cells into injured spinal cord and promotes functional recovery after compression-induced spinal cord injury in mice[J]. Brain Res, 2007, 1149: 223-231.
[27] 沈朝兰,李楚,朱晓波,等. 双孔钾离子通道激动剂利鲁唑对叔丁基过氧化氢诱导的人视网膜色素上皮细胞氧化损伤的作用[J]. 中华眼底病杂志, 2013, 29(4): 400-405.
[28] De Preter V.Metabonomics and systems biology[J]. Methods Mol Biol, 2015, 1277: 245-255.
[29] Taylor J, King RD, Altmann T, et al.Application of metabolomics to plant genotype discrimination using statistics and machine learning[J]. Bioinformatics, 2002, 18(Suppl 2): S241-S248.
[30] Kwon BK, Streijger F, Fallah N, et al.Cerebrospinal fluid biomarkers to stratify injury severity and predict outcome in human traumatic spinal cord injury[J]. J Neurotrauma, 2017, 34(3): 567-580.
[31] Pouw MH, Kwon BK, Verbeek MM, et al.Structural biomarkers in the cerebrospinal fluid within 24?h after a traumatic spinal cord injury: a descriptive analysis of 16 subjects[J]. Spinal Cord, 2014, 52(6): 428-433.
[32] Mori H, Hosoda K, Matsubara E, et al.Tau in cerebrospinal fluids: establishment of the sandwich ELISA with antibody specific to the repeat sequence in tau[J]. Neurosci Lett, 1995, 186(2-3): 181-183.
[33] Wolf H, Krall C, Pajenda G, et al.Alterations of the biomarker S-100B and NSE in patients with acute vertebral spine fractures[J]. Spine J, 2014, 14(12): 2918-2922.
[34] Shaw G, Yang C, Ellis R, et al.Hyperphosphorylated neurofilament NF-H is a serum biomarker of axonal injury[J]. Biochem Biophys Res Commun, 2005, 336(4): 1268-1277.
[35] Lubieniecka JM, Streijger F, Lee JH, et al.Biomarkers for severity of spinal cord injury in the cerebrospinal fluid of rats[J]. PLoS One, 2011, 6(4): e19247.
[36] Sengupta MB, Basu M, Iswarari S, et al.CSF proteomics of secondary phase spinal cord injury in human subjects: perturbed molecular pathways post injury[J]. PLoS One, 2014, 9(10): e110885.
[37] Xu D, Omura T, Masaki N, et al.Increased arachidonic acid-containing phosphatidylcholine is associated with reactive microglia and astrocytes in the spinal cord after peripheral nerve injury[J]. Sci Rep, 2016, 6: 26427.
[38] Kuhle J, Gaiottino J, Leppert D, et al.Serum neurofilament light chain is a biomarker of human spinal cord injury severity and outcome[J]. J Neurol Neurosurg Psychiatry, 2015, 86(3): 273-279.
[39] Vijayaprakash KM, Sridharan N.An experimental spinal cord injury rat model using customized impact device: a cost-effective approach[J]. J Pharmacol Pharmacother, 2013, 4(3): 211-213.
[40] McDonough A, Monterrubio A, Ariza J, et al. Calibrated forceps model of spinal cord compression injury [J]. J Vis Exp, 2015(98). doi: 10.3791/52318.
[41] Fouad K, Schnell L, Bunge MB, et al.Combining Schwann cell bridges and olfactory-ensheathing glia grafts with chondroitinase promotes locomotor recovery after complete transection of the spinal cord[J]. J Neurosci, 2005, 25(5): 1169-1178.
[42] Fujieda Y, Ueno S, Ogino R, et al.Metabolite profiles correlate closely with neurobehavioral function in experimental spinal cord injury in rats[J]. PLoS One, 2012, 7(8): e43152.
[43] Peng J, Zeng J, Cai B, et al.Establishment of quantitative severity evaluation model for spinal cord injury by metabolomic fingerprinting[J]. PLoS One, 2014, 9(4): e93736.