[1] Zagrebelsky M, Korte M. Maintaining stable memory engrams: new roles for Nogo-A in the CNS [J]. Neuroscience, 2014, 283: 17-25.
[2] Huber A B, Weinmann O, Br?samle C, et al. Patterns of Nogo mRNA and protein expression in the developing and adult rat and after CNS lesions [J]. J Neurosci, 2002, 22(9): 3553-3567.
[3] Schwab M E, Strittmatter S M. Nogo limits neural plasticity and recovery from injury [J]. Curr Opin Neurobiol, 2014, 27: 53-60.
[4] Voeltz G K, Prinz W A, Shibata Y, et al. A class of membrane proteins shaping the tubular endoplasmic reticulum [J]. Cell, 2006, 130(4): 573-586.
[5] Mcdonald C L, Bandtlow C, Reindl M. Targeting the Nogo receptor complex in diseases of the central nervous system [J]. Curr Med Chem, 2011, 18(2): 234-244.
[6] Sun Z, Dai X, Li Y, et al. A novel Nogo-66 receptor antagonist peptide promotes neurite regeneration in vitro [J]. Mol Cell Neurosci, 2016, 71: 80-91.
[7] Iobbi C, Korte M, Zagrebelsky M. Nogo-66 restricts synaptic strengthening via Lingo1 and the ROCK2-Cofilin pathway to control actin dynamics [J]. Cereb Cortex, 2017, 27(5): 2779-2792.
[8] Peng W S, Qi C, Zhang H, et al. Distribution of paired immunoglobulin-like receptor B in the nervous system related to regeneration dififculties after unilateral lumbar spinal cord injury [J]. Neural Regen Res, 2015, 10(7): 1139-1146.
[9] Kempf A, Tews B, Arzt M E, et al. The sphingolipid receptor S1PR2 is a receptor for Nogo-A repressing synaptic plasticity [J]. PLoS Biol, 2014, 12(1): e1001763.
[10] Caroni P, Donato F, Muller D. Structural plasticity upon learning: regulation and functions [J]. Nat Rev Neurosci, 2012, 13(7): 478-490.
[11] Toda T, Gage F H. Review: adult neurogenesis contributes to hippocampal plasticity [J]. Cell Tissue Res, 2018, 373(3): 693-709.
[12] Wills Z P, Mandelbrehm C, Mardinly A R, et al. The Nogo receptor family restricts synapse number in the developing hippocampus [J]. Neuron, 2012, 73(3): 466-481.
[13] Petrinovic M M, Hourez R, Aloy E M, et al. Neuronal Nogo-A negatively regulates dendritic morphology and synaptic transmission in the cerebellum [J]. Proc Natl Acad Sci U S A, 2013, 110(3): 1083-1088.
[14] Akbik F V, Bhagat S M, Patel P R, et al. Anatomical plasticity of adult brain is titrated by Nogo Receptor 1 [J]. Neuron, 2013, 77(5): 859-866.
[15] Zemmar A, Weinmann O, Kellner Y, et al. Neutralization of Nogo-A enhances synaptic plasticity in the rodent motor cortex and improves motor learning in vivo [J]. J Neurosci, 2014, 34(26): 8685-8698.
[16] Guzik-Kornacka A, van der Bouga A, Vajda F, et al. Nogo-A deletion increases the plasticity of the optokinetic response and changes retinal projection organization in the adult mouse visual system [J]. Brain Struct Funct, 2016, 221(1): 1-13.
[17] Djurisic M, Vidal G S, Mann M, et al. PirB regulates a structural substrate for cortical plasticity [J]. Proc Natl Acad Sci U S A, 2013, 110(51): 20771-20776.
[18] Tews B, Kai S, Arzt M E, et al. Synthetic microRNA-mediated downregulation of Nogo-A in transgenic rats reveals its role as regulator of synaptic plasticity and cognitive function [J]. Proc Natl Acad Sci U S A, 2013, 110(16): 6583-6588.
[19] Lee H, Raiker S J, Venkatesh K, et al. Synaptic function for the Nogo-66 receptor NgR1: regulation of dendritic spine morphology and activity-dependent synaptic strength [J]. J Neurosci, 2008, 28(11): 2753-2765.
[20] Meeker R, Williams K. Dynamic nature of the p75 neurotrophin receptor in response to injury and disease [J]. J Neuroimmune Pharmacol, 2014, 9(5): 615-628.
[21] Raiker S J, Lee H, Baldwin K T, et al. Oligodendrocyte-myelin glycoprotein and Nogo negatively regulate activity-dependent synaptic plasticity [J]. J Neurosci, 2010, 30(37): 12432-12445.
[22] Joset A, Dodd D A, Halegoua S, et al. Pincher-generated Nogo-A endosomes mediate growth cone collapse and retrograde signaling [J]. J Cell Biol, 2010, 188(2): 271-285.
[23] Tong J, Liu W, Wang X, et al. Inhibition of Nogo-66 receptor 1 enhances recovery of cognitive function after traumatic brain injury in mice [J]. J Neurotrauma, 2013, 30(4): 247-258.
[24] Karlsson T E, Smedfors G, Brodin A T, et al. NgR1: a tunable sensor regulating memory formation, synaptic, and dendritic plasticity [J]. Cereb Cortex, 2016, 26(4): 1804-1817.
[25] Gu H, Yu S P, Gutekunst C A, et al. Inhibition of the Rho signaling pathway improves neurite outgrowth and neuronal differentiation of mouse neural stem cells [J]. Int J Physiol Pathophysiol Pharmacol, 2013, 5(1): 11-20.
[26] Wu B Q, Bi Z G, Qi Q. Inactivation of the Rho-ROCK signaling pathway to promote neurologic recovery after spinal cord injuries in rats [J]. Chin Med J (Engl), 2013, 126(19): 3723-3727.
[27] Chaudhuri A. Multiple sclerosis is primarily a neurodegenerative disease [J]. J Neural Transm (Vienna), 2013, 120(10): 1463-1466.
[28] Petratos S, Ozturk E, Azari M F, et al. Limiting multiple sclerosis related axonopathy by blocking Nogo receptor and CRMP-2 phosphorylation [J]. Brain, 2012, 135(Pt 6): 1794-1818.
[29] Yang Y, Liu Y, Wei P, et al. Silencing Nogo-A promotes functional recovery in demyelinating disease [J]. Ann Neurol, 2010, 67(4): 498-507.
[30] Ineichen B V, Kapitza S, Bleul C, et al. Nogo-A antibodies enhance axonal repair and remyelination in neuro-inflammatory and demyelinating pathology [J]. Acta Neuropathol, 2017, 134(3): 423-440.
[31] Li X, Zhang Y, Yan Y, et al. LINGO-1-Fc-transduced neural stem cells are effective therapy for chronic stage experimental autoimmune encephalomyelitis [J]. Mol Neurobiol, 2017, 54(6): 4365-4378.
[32] Gil V, Nicolas O, Mingorance A, et al. Nogo-A expression in the human hippocampus in normal aging and in Alzheimer disease [J]. J Neuropathol Exp Neurol, 2006, 65(5): 433-444.
[33] Zhou X D, Hu X Y, He W X, et al. Interaction between amyloid precursor protein and Nogo receptors regulates amyloid deposition [J]. FASEB J, 2011, 25(9): 3146-3156.
[34] Zhu H Y, Guo H F, Hou H L, et al. Increased expression of the Nogo receptor in the hippocampus and its relation to the neuropathology in Alzheimer's disease [J]. Hum Pathol, 2007, 38(3): 426-434.
[35] Kim T, Vidal G S, Djurisic M, et al. Human lilrB2 is a β-amyloid receptor and its murine homolog PirB regulates synaptic plasticity in an Alzheimer's model [J]. Science, 2013, 341(6152): 1399-1404.
[36] Asle-Rousta M, Oryan S, Ahmadiani A, et al. Activation of sphingosine 1-phosphate receptor-1 by SEW2871 improves cognitive function in Alzheimer's disease model rats [J]. EXCLI J, 2013, 12: 449-461.
[37] Schroeder B W, Shinnick-Gallagher P. Fear memories induce a switch in stimulus response and signaling mechanisms for long-term potentiation in the lateral amygdale [J]. Eur J Neurosci, 2015, 20(2): 549-556.
[38] Huang C C, Chen C C, Liang Y C, et al. Long-term potentiation at excitatory synaptic inputs to the intercalated cell masses of the amygdale [J]. Int J Neuropsychopharmacol, 2014, 17(8): 1233-1242.
[39] Collingridge G L, Peineau S, Howland J G, et al. Long-term depression in the CNS [J]. Nat Rev Neurosci, 2010, 11(7): 459-473.
[40] Dong Z F, Bai Y R, Wu X Y, et al. Hippocampal long-term depression mediates spatial reversal learning in the Morris water maze [J]. Neuropharmacology, 2013, 64: 65-73.
[41] Bhagat S M, Butler S S, Taylor J R, et al. Erasure of fear memories is prevented by Nogo receptor 1 in adulthood [J]. Mol Psychiatry, 2016, 21(9): 1281-1289.
