[1] Stead LG, Bodhit AN, Patel PS, et al. TBI surveillance using the common data elements for traumatic brain injury: a population study [J]. Int J Emerg Med, 2013, 6(1): 5-11. [2] Russo MV, McGavern DB. Inflammatory neuroprotection following traumatic brain injury [J]. Science, 2016, 353(6301): 783-785. [3] Dalgard CL, Cole JT, Kean WS, et al. The cytokine temporal profile in rat cortex after controlled cortical impact [J]. Front Mol Neurosci, 2012, 5: 6. [4] Liu S, Zhang L, Wu Q, et al. Chemokine CCL2 induces apoptosis in cortex following traumatic brain injury [J]. J Mol Neurosci, 2013, 51(3): 1021-1029. [5] Chiu CC, Liao YE, Yang LY, et al. Neuroinflammation in animal models of traumatic brain injury [J]. J Neurosci Methods, 2016, 272: 38-49. [6] Perez-Polo JR, Rea HC, Johnson KM, et al. Inflammatory consequences in a rodent model of mild traumatic brain injury [J]. J Neurotrauma, 2013, 30(9): 727-740. [7] Lo U, Selvaraj V, Plane JM, et al. P38a (MAPK14) critically regulates the immunological response and the production of specific cytokines and chemokines in astrocytes [J]. Sci Rep, 2014, 4: 7405-7408. [8] Johnson EA, Dao TL, Guignet MA, et al. Increased expression of the chemokines CXCL1 and MIP-1α by resident brain cells precedes neutrophil infiltration in the brain following prolonged soman-induced status epilepticus in rats [J]. J Neuroinflammation, 2011, 8: 41-50. [9] Rumble JM, Huber AK, Krishnamoorthy G. Neutrophil-related factors as biomarkers in EAE and MS [J]. J Exp Med, 2015, 212(1): 23-35. [10] Marro BS, Grist JJ, Lane TE. Inducible expression of CXCL1 within the central nervous system amplifies viral-induced demyelination [J]. J Immunol, 2016, 196(4): 1855-1864. [11] Ellman DG, Degn M, Lund MC, et al. Genetic ablation of soluble TNF does not affect lesion size and functional recovery after moderate spinal cord injury in mice [J]. Mediators Inflamm, 2016, 2016: 2684098. doi: 10.1155/2016/2684098. [12] Remus EW, Sayeed I, Won S. Progesterone protects endothelial cells after cerebrovascular occlusion by decreasing MCP-1- and CXCL1-mediated macrophage infiltration [J]. Exp Neurol, 2015, 271: 401-408. [13] Szmydynger-Chodobska J, Shan R, Thomasian N, et al. The involvement of pial microvessels in leukocyte invasion after mild traumatic brain injury [J]. PLoS One, 2016, 11(12): e0167677. [14] Joanna SC, Nathalie S, Brian JZ, et al. The role of the choroid plexus in neutrophil invasion after traumatic brain injury [J]. Cereb Blood Flow Metab, 2009, 29(9): 1503-1516. [15] Semple BD, Bye N, Ziebell JM, et al. Deficiency of the chemokine receptor CXCR2 attenuates neutrophil infiltration and cortical damage following closed head injury [J]. Neurobiol Dis, 2010, 40(2): 394-403. |