[1] Davidoff GN, Roth EJ, Richards JS. Cognitive deficits in spinal cord injury: epidemiology and outcome [J]. Arch Phys Med Rehabil, 1992, 73(3): 275-284. [2] Davidoff G, Morris J, Roth E, et al. Cognitive dysfunction and mild closed head injury in traumatic spinal cord injury [J]. Arch Phys Med Rehabil, 1985, 66(8): 489-491. [3] Wilmot CB, Cope DN, Hall KM, et al. Occult head injury: its incidence in spinal cord injury [J]. Arch Phys Med Rehabil, 1985, 66(4): 227-231. [4] Richards JS, Brown L, Hagglund K, et al. Spinal cord injury and concomitant traumatic brain injury. Results of a longitudinal investigation [J]. Am J Phys Med Rehabil, 1988, 67(5): 211-216. [5] Roth E, Davidoff G, Thomas P, et al. A controlled study of neuropsychological deficits in acute spinal cord injury patients [J]. Paraplegia, 1989, 27(6): 480-489. [6] Lazzaro I, Tran Y, Wijesuriya N, et al. Central correlates of impaired information processing in people with spinal cord injury [J]. J Clin Neurophysiol, 2013, 30(1): 59-65. [7] Di Russo F, Bultrini A, Brunelli S, et al. Benefits of sports participation for executive function in disabled athletes [J]. J Neurotrauma, 2010, 27(12): 2309-2319. [8] Gordon E. Neuroimaging in neuropsychiatry [J]. Semin Clin Neuropsychiatry, 2002, 7(1): 42-53. [9] Noriega DB, Savelkoul HF. Immune dysregulation in autism spectrum disorder [J]. Eur J Pediatr, 2014, 173(1): 33-43. [10] Rivat C, Becker C, Blugeot A, et al. Chronic stress induces transient spinal neuroinflammation, triggering sensory hypersensitivity and long-lasting anxiety-induced hyperalgesia [J]. Pain, 2010, 150(2): 358-368. [11] Widerstr?m-Noga EG, Felipe-Cuervo E, Broton JG, et al. Perceived difficulty in dealing with consequences of spinal cord injury [J]. Arch Phys Med Rehabil, 1999, 80(5): 580-586. [12] Murray RF, Asghari A, Egorov DD, et al. Impact of spinal cord injury on self-perceived pre- and postmorbid cognitive, emotional and physical functioning [J]. Spinal Cord, 2007, 45(6): 429-436. [13] Hess DW, Marwitz JH, Kreutzer JS. Neuropsychological impairments after spinal cord injury: a comparative study with mild traumatic brain injury [J]. Rehabil Psychol, 2003, 48(3): 151-156. [14] Davidoff G, Morris J, Roth E, et al. Closed head injury in spinal cord injured patients: retrospective study of loss of consciousness and post-traumatic amnesia [J]. Arch Phys Med Rehabil, 1985, 66(1): 41-43. [15] Davidoff G, Thomas P, Johnson M, et al. Closed head injury in acute traumatic spinal cord injury: incidence and risk factors [J]. Arch Phys Med Rehabil, 1988, 69(10): 869-872. [16] Hovda DA. The neurophysiology of concussion [J]. Prog Neurol Surg, 2014, 28: 28-37. [17] Wang Q, Wang Z, Zhu P, et al. Alterations of myelin basic protein and ultrastructure in the limbic system at the early stage of trauma-related stress disorder in dogs [J]. J Trauma, 2004, 56(3): 604-610. [18] Zhang B, Huang Y, Su Z, et al. Neurological, functional, and biomechanical characteristics after high-velocity behind armor blunt trauma of the spine [J]. J Trauma, 2011, 71(6): 1680-1688. [19] 冯东亮,南伟,伍亚民,等. 脊髓损伤大鼠的学习记忆功能与海马病理改变[J]. 中国康复理论与实践, 2015, 21(11): 1267-1272. [20] Freund P, Weiskopf N, Ashburner J, et al. MRI investigation of the sensorimotor cortex and the corticospinal tract after acute spinal cord injury: a prospective longitudinal study [J]. Lancet Neurol, 2013, 12(9): 873-881. [21] 侯景明. 脊髓损伤后脑结构和功能改变的多模态磁共振研究[D]. 重庆:第三军医大学, 2015. [22] Nicotra A, Critchley HD, Mathias CJ, et al. Emotional and autonomic consequences of spinal cord injury explored using functional brain imaging [J]. Brain, 2006, 129(Pt 3): 718-728. [23] Hilton BJ, Anenberg E, Harrison TC, et al. Re-establishment of cortical motor output maps and spontaneous functional recovery via spared dorsolaterally projecting corticospinal neurons after dorsal column spinal cord injury in adult mice [J]. J Neurosci, 2016, 36(14): 4080-4092. [24] Han Q, Cao C, Ding Y, et al. Plasticity of motor network and function in the absence of corticospinal projection [J]. Exp Neurol, 2015, 267: 194-208. [25] Hains BC, Black JA, Waxman SG. Primary cortical motor neurons undergo apoptosis after axotomizing spinal cord injury [J]. J Comp Neurol, 2003, 462(3): 328-341. [26] Fumagalli F, Madaschi L, Caffino L, et al. Acute spinal cord injury reduces brain derived neurotrohic factor expression in rat hippocampus [J]. Neuroscience, 2009, 159(3): 936-939. [27] Chang CM, Lee MH, Wang TC, et al. Brain protection by methylprednisolone in rats with spinal cord injury [J]. Neuroreport, 2009, 20(10): 968-972. [28] Wu J, Stoica BA, Luo T, et al. Isolated spinal cord contusion in rats induces chronic brain neuroinflammation, neurodegeneration, and cognitive impairment. Involvement of cell cycle activation [J]. Cell Cycle, 2014, 13(15): 2446-2458. [29] Wu J, Raver C, Piao C, et al. Cell cycle activation contributes to increased neuronal activity in the posterior thalamic nucleus and associated chronic hyperesthesia after rat spinal cord contusion [J]. Neurotherapeutics, 2013, 10(3): 520-538. [30] Guerrero AR, Uchida K, Nakajima H, et al. Blockade of interleukin-6 signaling inhibits the classic pathway and promotes an alternative pathway of macrophage activation after spinal cord injury in mice [J]. J Neuroinflammation, 2012, 9: 40. [31] Kipnis J, Gadani S, Derecki NC. Pro-cognitive properties of T cells [J]. Nat Rev Immunol, 2012, 12(9): 663-669. [32] Skaper SD, Facci L, Giusti P. Neuroinflammation, microglia and mast cells in the pathophysiology of neurocognitive disorders: a review [J]. CNS Neurol Disord Drug Targets, 2014, 13(10): 1654-1666. [33] Hotamisligil GS. Endoplasmic reticulum stress and the inflammatory basis of metabolic disease [J]. Cell, 2010, 140(6): 900-917. [34] Wu J, Zhao Z, Kumar A, et al. Endoplasmic reticulum stress and disrupted neurogenesis in the brain are associated with cognitive impairment and depressive-like behavior after spinal cord injury [J]. J Neurotrauma, 2016 33(21): 1919-1935. [35] Claydon VE, Krassioukov AV. Orthostatic hypotension and autonomic pathways after spinal cord injury [J]. J Neurotrauma, 2006, 23(12): 1713-1725. [36] Phillips AA, Warburton DE, Ainslie PN, et al. Regional neurovascular coupling and cognitive performance in those with low blood pressure secondary to high-level spinal cord injury: improved by alpha-1 agonist midodrine hydrochloride [J]. J Cereb Blood Flow Metab, 2014, 34(5): 794-801. |