[1] Winstein CJ, Stein J, Arena R, et al.Guidelines for adult stroke rehabilitation and recovery: a guideline for health care professionals from the American Heart Association/American Stroke Association[J]. Stroke, 2016, 47(6): e98-e169. [2] 唐强,朱路文. 脑卒中康复新策略——针康法[J]. 中国康复医学杂志, 2015, 30(10): 1071-1073. [3] 唐强,姜云飞,朱路文,等. 针康法对局灶性脑缺血大鼠神经功能缺损及缺血区碱性成纤维细胞生长因子、血管抑制素蛋白表达的影响[J]. 中国康复理论与实践, 2015, 21(10): 1151-1155. [4] 唐强,田源,叶涛,等. 针康法对脑缺血大鼠缺血半暗区细胞凋亡及X连锁凋亡抑制蛋白和cleaved-caspase-9蛋白表达的影响[J]. 中国康复理论与实践, 2017, 23(12): 1365-1371. [5] 唐强,叶涛,朱路文,等. 针康法对脑缺血大鼠神经功能和细胞外信号调节激酶1/2信号通路的影响[J]. 中国康复理论与实践, 2017, 23(1): 27-31. [6] 叶涛,唐强,朱路文,等. 针康法对脑缺血大鼠神经功能和缺血半暗区皮层血管内皮生长因子受体表达的影响[J]. 中国康复理论与实践, 2017, 23(5): 520-524. [7] 朱路文,唐强,王艳,等. 针康法对脑缺血后神经血管单元重塑的影响[J]. 针灸临床杂志, 2016, 32(6): 70-73. [8] 唐强,叶涛,朱路文,等. 针康法对局灶性脑梗死大鼠神经功能缺损及梗死灶周边区RhoA、ROCK-Ⅱ蛋白表达的影响[J]. 针灸临床杂志, 2016, 32(8): 73-77. [9] 唐强,朱路文,邢艳丽,等. 针康法对脑缺血大鼠运动功能及GAP-43、Nogo-A表达的影响[J]. 针灸临床杂志, 2015, 31(1): 54-57. [10] 毛德文,陈月桥,王丽,等. Caspase-8及Caspase-3与细胞凋亡[J]. 辽宁中医药大学学报, 2008, 10(10): 148-150. [11] Mohamed MS, Bishr MK, Almutairi FM, et al.Inhibitors of apoptosis: clinical implications in cancer[J]. Apoptosis, 2017, 22(12): 1487-1509. [12] Estornes Y, Bertrand MJ.IAPs, regulators of innate immunity and inflammation[J]. Semin Cell Dev Biol, 2015, 39(3): 106-114. [13] Varfolomeev E, Goncharov T, Vucic D.Roles of c-IAP proteins in TNF receptor family activation of NF-κB signaling [M]// May M. NF-kappa B. Methods in Molecular Biology. New York: Humana Press, 2015: 269-282. [14] Koizumi J.Experimental studies of ischemic brain edema. 1. A new experimental model of cerebral embolism in rats in which recirculation can be introduced in the ischemic area[J]. Jpn J stroke, 1986, 16(8): 1-8. [15] Tsai LK, Wang Z, Munasinghe J, et al.Mesenchymal stem cells primed with valproate and lithium robustly migrate to infarcted regions and facilitate recovery in a stroke model[J]. Stroke, 2011, 42(10): 2932-2939. [16] 林冰冰,王鲜,柳维林,等. 弥散张量成像观察电针对缺血性脑卒中大鼠运动皮层-纹状体神经传导束的影响[J]. 中国康复理论与实践, 2017, 23(7): 756-761. [17] Zhang QW, Deng XX, Sun X, et al.Exercise promotes axon regeneration of newborn striatonigral and corticonigral projection neurons in rats after ischemic stroke[J]. PLoS One, 2013, 8(11): e80139. [18] Chen J, Sanberg PR, Li Y, et al.Intravenous administration of human umbilical cord blood reduces behavioral deficits after stroke in rats[J]. Stroke, 2001, 32(11): 2682-2688. [19] 李花,刘旺华,周小青,等. 活血通络组方对大鼠脑缺血再灌注损伤后神经功能恢复及其抗凋亡机制研究[J]. 中国康复医学杂志, 2009, 24(10): 880-883. [20] 夏相宜,肖长江,王宇红,等. 脑健胶囊对MCAO大鼠脑组织Fas凋亡途径的影响[J]. 湖南中医药大学学报, 2015, 35(1): 11-15. [21] 赵瑞杰,李引乾,王会,等. Caspase家族与细胞凋亡的关系[J]. 中国畜牧杂志, 2010, 46(17): 73-78. [22] Pfeffer CM, Singh AT.Apoptosis: a target for anticancer therapy[J]. Int J Mol Sci, 2018, 19(2): E448. [23] Gyrd-Hansen M, Meier P.IAPs: from caspase inhibitors to modulators of NF-κB, inflammation and cancer[J]. Nat Rev Cancer, 2010, 10(8): 561-574. [24] Dubrez-Daloz L, Dupoux A, Cartier J.IAPs: more than just inhibitors of apoptosis proteins[J]. Cell Cycle, 2008, 7(8): 1036-1046. [25] Deveraux QL, Roy N, Stennicke HR, et al.IAPs block apoptotic events induced by caspase-8 and cytochrome C by direct inhibition of distinct caspases[J]. EMBO J, 1998, 17(8): 2215-2223. [26] Eckelman BP, Salvesen GS.The human anti-apoptotic proteins cIAP1 and cIAP2 bind but do not inhibit caspases[J]. J Biol Chem, 2006, 281(6): 3254. [27] Figiel I.Pro-inflammatory cytokine TNF-alpha as a neuroprotective agent in the brain[J]. Acta Neurobiol Exp (Wars), 2008, 68(4): 526-534. [28] Berthelet J, Dubrez L.Regulation of apoptosis by inhibitors of apoptosis (IAPs)[J]. Cells, 2013, 2(1): 163-187. [29] Hu S, Yang X.Cellular inhibitor of apoptosis 1 and 2 are ubiquitin ligases for the apoptosis inducer Smac/DIABLO[J]. J Biol Chem, 2003, 278(12): 10055-10060. [30] Cheung HH, Plenchette S, Kern CJ, et al.The RING domain of cIAP1 mediates the degradation of RING-bearing inhibitor of apoptosis proteins by distinct pathways[J]. Mol Biol Cell, 2008, 19(7): 2729-2740. [31] Feltham RL, Moulin M, Vince JE, et al.Tumor necrosis factor (TNF) signaling, but not TWEAK (TNF-like weak inducer of apoptosis)-triggered cIAP1 (cellular inhibitor of apoptosis protein 1) degradation, requires cIAP1 RING dimerization and E2 binding[J]. J Biol Chem, 2017, 292(34): 14310. |