1 KatanM, LuftA. Global burden of stroke [J]. Semin Neurol, 2018, 38(2): 208-211. 2 ThusharaV N, SangwanA, SharmaB, et al. Cerebral ischemic preconditioning: the road so far… [J]. Mol Neurobiol, 2016, 53(4): 2579-2593. 3 HuberR, KasischkeK, LudolphA C, et al. Increase of cellular hypoxic tolerance by erythromycin and other antibiotics [J]. Neuroreport, 1999, 10(7): 1543-1546. 4 KitagawaK. Ischemic tolerance in the brain: endogenous adaptive machinery against ischemic stress [J]. J Neurosci Res, 2012, 90(5): 1043-1054. 5 邱龙,周爽,尤艳利. “治未病”思想在针灸预防中风中的运用及机理研究概况[J]. 中国中医药信息杂志, 2009, 16(8): 100-102. 6 章薇. 针灸——在中医“治未病”的引领作用中占主导地位[J]. 中国针灸, 2018, 38(11): 1218. 7 LiuA J, LiJ H, LiH Q, et al. Electroacupuncture for acute ischemic stroke: a meta-analysis of randomized controlled trials [J]. Am J Chin Med, 2015, 43(8): 1541-1566. 8 XiongL Z, LuZ H, HouL C, et al. Pretreatment with repeated electroacupuncture attenuates transient focal cerebral ischemic injury in rats [J]. Chin Med J, 2003, 116(1): 108-111. 9 BariM, BattistaN, FezzaF, et al. New insights into endocannabinoid degradation and its therapeutic potential [J]. Mini Rev Med Chem, 2006, 6(3): 257-268. 10 GalveR I, AguadoT, PalazuelosJ, et al. Mechanisms of control of neuron survival by the endocannabinoid system [J]. Curr Pharm Des, 2008, 14(23): 2279-2288. 11 CapettiniL S, SavergniniS Q, DaS R, et al. Update on the role of cannabinoid receptors after ischemic stroke [J]. Mediators Inflamm, 2012, 2012: 1-8. 12 ZhangM, MartinB R, AdlerM W, et al. Cannabinoid CB(2) receptor activation decreases cerebral infarction in a mouse focal ischemia/reperfusion model [J]. J Cere Blood Flow Metab, 2007, 27(7): 1387-1396. 13 马磊. 大麻素受体参与电针预处理诱导不同时相的脑保护作用[D]. 西安:第四军医大学, 2010. 14 WangQ, PengY, ChenS, et al. Pretreatment with electroacupuncture induces rapid tolerance to focal cerebral ischemia through regulation of endocannabinoid system [J]. Stroke, 2009, 40(6): 2157-2164. 15 WangQ, LiX Y, ChenY H, et al. Activation of epsilon protein kinase C-mediated anti-apoptosis is involved in rapid tolerance induced by electroacupuncture pretreatment through cannabinoid receptor type 1 [J]. Stroke, 2011, 42(2): 389-396. 16 DuJ, WangQ, HuB, et al. Involvement of ERK1/2 activation in electroacupuncture pretreatment via cannabinoid CB1 receptor in rats [J]. Brain Res, 2010, 1360: 1-7. 17 WeiH D, YaoX, YangL F, et al. Glycogen synthase kinase-3β is involved in electroacupuncture pretreatment via the cannabinoid CB1 receptor in ischemic stroke [J]. Mol Neurobiol, 2014, 49(1): 326-336. 18 ZhouH, ZhangZ, WeiH D, et al. Activation of STAT3 is involved in neuroprotection by electroacupuncture pretreatment via cannabinoid CB1 receptors in rats [J]. Brain Res, 2013, 1529: 154-164. 19 MayorD, TymianskiM. Neurotransmitters in the mediation of cerebral ischemic injury [J]. Neuropharmacology, 2017, 134: 178-188. 20 TakahashiK, FosterJ B, LinC L. Glutamate transporter EAAT2: regulation, function, and potential as a therapeutic target for neurological and psychiatric disease [J]. Cell Mol Life Sci, 2015, 72(18): 3489-3506. 21 朱爱,王海云. γ-氨基丁酸及其受体在脑缺血性损伤中的作用[J]. 国际麻醉学与复苏杂志, 2013, 34(11): 1047-1050. 22 陈巍,张森森,孙彭龄. 电针预处理对大鼠脑缺血再灌注损伤后EAAT2的表达研究[J]. 中华神经外科疾病研究杂志, 2014, 13(3): 232-235. 23 GuoZ Z, ZhangL, WuY, et al. The role of glutamate transporter-1 in the acquisition of brain ischaemic tolerance in rats induced by electro-acupuncture pre-treatment [J]. Brain Inj, 2015, 29(3): 396-402. 24 王枫. 电针预处理通过EAAT2调节NDRG2诱导脑缺血耐受的研究[D]. 西安:第四军医大学, 2012. 25 郭真真,吴毅,贾杰,等. 电针预处理对脑缺血再灌注损伤大鼠谷氨酸和γ-氨基丁酸的影响[J]. 实用老年医学, 2013, 27(3): 202-207. 26 LiJ, O W, LiW, et al. Oxidative stress and neurodegenerative disorders [J]. Int J Mol Sci, 2013, 14(12): 24438-24475. 27 SunS S, ChenX Y, GaoY, et al. Mn-SOD upregulation by electroacupuncture attenuates ischemic oxidative damage via CB1R-mediated STAT3 phosphorylation [J]. Mol Neurobiol, 2016, 53(1): 331-343. 28 姜蓓蓓,谭丽,姚辉,等. Peroxiredoxin 6在脑缺血再灌注损伤中的抗氧化和抗凋亡作用[J]. 重庆医科大学学报, 2016, 41(8): 781-786. 29 高杨,孙思斯,刘曌宇,等. 电针预处理通过上调Peroxiredoxin 6减轻大鼠脑缺血再灌注损伤[J]. 神经解剖学杂志, 2014, 30(5): 507-513. 30 HotamisligilG S. Endoplasmic reticulum stress and the inflammatory basis of metabolic disease [J]. Cell, 2010, 140(6): 900-917. 31 SuY C, LiF. Endoplasmic reticulum stress in brain ischemia [J]. Int J Neurosci, 2016, 126(8): 681-691. 32 AvilaM F, CabezasR, TorrenteD, et al. Novel interactions of GRP78: UPR and estrogen responses in the brain [J]. Cell Biol Int, 2013, 37(6): 521-532. 33 陈怀龙,齐慧,刘孝洁,等. 电针预处理对全脑缺血再灌注损伤大鼠海马葡萄糖调节蛋白78和生长停滞及DNA损伤基因153表达的影响[J]. 针刺研究, 2014, 39(6): 431-436. 34 屈生彪,王勇. 内质网应激在脑缺血再灌注损伤中作用[J]. 华夏医学, 2018, 31(1): 170-174. 35 肖飞,张高峰,尹曾,等. 电针预处理对脑缺血再灌注大鼠皮层内质网IRE1-XBP1信号通路的影响[J]. 中华麻醉学杂志, 2018, 38(2): 226-229. 36 JinR, LiuL, ZhangS, et al. Role of inflammation and its mediators in acute ischemic stroke [J]. J Cardiovasc Transl Res, 2013, 6(5): 834-851. 37 朱路文,叶涛,姜云飞,等. 电针预处理对大鼠脑缺血再灌注损伤后炎性因子及细胞凋亡的影响[J]. 中国康复理论与实践, 2016, 22(7): 765-768. 38 金深辉,刘俊伟,罗亮,等. 电针预处理对脑缺血再灌注大鼠皮质及纹状体IL-1β表达的影响[J]. 浙江中医杂志, 2012, 47(4): 255-256. 39 叶涛,朱路文,唐强,等. 电针预处理对大鼠脑缺血再灌注损伤后脑梗死体积及血清TNF-α、IL-10含量的影响[J]. 中国针灸, 2017, 37(10): 1093-1098. 40 叶涛,朱路文,唐强,等. 电针预处理对脑缺血再灌注损伤大鼠神经功能和缺血半暗区Tolls样受体4、核转录因子κB蛋白的影响[J]. 中国康复理论与实践, 2017, 23(7): 745-749. 41 牟传琳,王燕,李文洁,等. 电针预处理对小鼠脑缺血再灌注时海马IκB-α∕NF-κB∕ICAM-1信号通路的影响[J]. 中华麻醉学杂志, 2018, 38(8): 1012-1016. 42 马治,白福海,周恒,等. 胆碱能抗炎通路在电针预处理调控脑缺血再灌注大鼠小胶质细胞活化状态中的作用[J]. 神经解剖学杂志, 2016, 32(6): 745-750. 43 WangQ, WangF, LiX, et al. Electroacupuncture pretreatment attenuates cerebral ischemic injury through α7 nicotinic acetylcholine receptor-mediated inhibition of high-mobility group box 1 release in rats [J]. J Neuroinflammation, 2012, 9: 24. 44 杨蕾,翟茜,江涛,等. 电针预处理对脑缺血再灌注后小胶质细胞活化状态的影响[J]. 现代生物医学进展, 2015, 15(20): 3826-3829. 45 LiY C, WeiZ, JiangZ, et al. New progress in the approaches for blood-brain barrier protection in acute ischemic stroke [J]. Brain Res Bull, 2019, 144: 46-57. 46 MenezesM J, McclenahanF K, LeitonC V, et al. The extracellular matrix protein laminin α2 regulates the maturation and function of the blood-brain barrier [J]. J Neurosci, 2014, 34(46): 15260-15280. 47 SifatA E, VaidyaB, AbbruscatoT J. Blood-brain barrier protection as a therapeutic strategy for acute ischemic stroke [J]. AAPS J, 2017, 19(4): 957-972. 48 邢东,吴志新,董辉,等. 电针预处理对MCAO大鼠模型血脑屏障和MMP-9的影响[J]. 神经解剖学杂志, 2017, 33(4): 383-389. 49 JungY S, LeeS W, ParkJ H, et al. Electroacupuncture preconditioning reduces ROS generation with NOX4 down-regulation and ameliorates blood-brain barrier disruption after ischemic stroke [J]. J Biomed Sci, 2016, 23(1): 32. 50 唐强,阮野,李宏玉,等. 运动或电针预处理对脑梗死大鼠神经功能缺损及层粘连蛋白表达的影响 [J]. 中国康复理论与实践, 2017, 23(3): 288-291. 51 林咸明,陈丽萍,姚旭. 不同时程电针预处理对脑缺血再灌注大鼠血脑屏障基质金属蛋白酶-9、血管内皮生长因子的影响[J]. 针刺研究, 2015, 40(1): 40-44. 52 林咸明,李金霞,姚旭. 不同时程电针预处理对脑缺血再灌注大鼠血脑屏障水通道蛋白-4及紧密连接蛋白-5的影响[J]. 中华物理医学与康复杂志, 2015, 37(7): 498-502. 53 刘建勋,林咸明. 不同时程电针预处理对脑缺血大鼠血脑屏障功能的保护效应[J]. 上海针灸杂志, 2014, 33(12): 1169-1172. 54 张亚敏,孙华,徐虹,等. 电针百会、足三里穴对脑缺血再灌注大鼠凋亡相关因子Bcl-2和Bax表达的影响[J]. 针灸临床杂志, 2017, 33(2): 37-40. 55 徐浩,沈剑,赵昱,等. 电针预处理大鼠百会穴对脑缺血保护作用及HIF-1α相关机制的研究[J]. 神经解剖学杂志, 2015, 31(5): 617-622. 56 叶涛,朱路文,唐强,等. 电针预处理对脑缺血再灌注大鼠缺血半暗区细胞凋亡及凋亡相关蛋白表达的影响[J]. 中国康复理论与实践, 2018, 24(1): 54-59. 57 HeX, MoY C, GengW J, et al. Role of Wnt/β-catenin in the tolerance to focal cerebral ischemia induced by electroacupuncture pretreatment [J]. Neurochem Int, 2016, 97: 124-132. 58 张照亮,刘玉秋,张高峰,等. 电针预处理对大鼠脑缺血再灌注时脑组织动力相关蛋白1活性的影响[J]. 中华麻醉学杂志, 2017, 37(12): 1498-1501. 59 WangP, ShaoB Z, DengZ Q, et al. Autophagy in ischemic stroke [J]. Prog Neurobiol, 2018, 163: 98-117. 60 WuZ Q, CuiS Y, ZhuL, et al. Study on the mechanism of mTOR-mediated autophagy during electroacupuncture pretreatment against cerebral ischemic injury [J]. Evid Based Complementray Alternat Med, 2016, 2016(5): 1-8. 61 WuZ, ZouZ, ZouR, et al. Electroacupuncture pretreatment induces tolerance against cerebral ischemia/reperfusion injury through inhibition of the autophagy pathway [J]. Mol Med Rep, 2015, 11(6): 4438-4446. 62 周文婷,陈怀龙,王明山,等. 电针预处理对脑缺血/再灌注小鼠海马mTOR表达的影响[J]. 中华临床医师杂志(电子版), 2016, 10(13): 1937-1941. |