[1] Bongers KS, Fox DK, Ebert SM, et al.Skeletal muscle denervation causes skeletal muscle atrophy through a pathway that involves both Gadd45a and HDAC4[J]. Am J Physiol Endocrinol Metab, 2013, 305(7): 907-915.
[2] Bonaldo P, Sandri M.Cellular and molecular mechanisms of muscle atrophy[J]. Dis Model Mech, 2013, 6(1): 25-39.
[3] 赵丹丹,唐成林,黄思琴,等.失神经性肌萎缩动物模型制备方法的研究进展[J]. 中国康复理论与实践, 2018, 24(1): 80-83.
[4] Léger B, Cartoni R, Praz M, et al.Akt signalling through GSK-3beta, mTOR and Foxo1 is involved in human skeletal muscle hypertrophy and atrophy[J]. J Physiol, 2006, 576(3): 923-933.
[5] Bodine SC, Baehr LM.Skeletal muscle atrophy and the E3 ubiquitin ligases MuRF1 and MAFbx/atrogin-1[J]. Am J Physiol Endocrinol Metab, 2014, 307(6): e469-e484.
[6] Zheng B, Ohkawa S, Li H, et al.FOXO3a mediates signaling crosstalk that coordinates ubiquitin and atrogin-1/MAFbx expression during glucocorticoid-induced skeletal muscle atrophy[J]. FASEB J, 2010, 24(8): 2660-2669.
[7] Cohen S, Nathan JA, Goldberg AL.Muscle wasting in disease: molecular mechanisms and promising therapies[J]. Nat Rev Drug Discov, 2015, 14(1): 58-74.
[8] 连晓阳,陈少清,方忆生,等. 电针三阴交、阴陵泉对腓肠肌损伤大鼠损伤后修复及生长因子表达的影响[J]. 中国康复理论与实践, 2015, 21(11): 1273-1276.
[9] Su Z, Hu L, Cheng J, et al.Acupuncture plus low-frequency electrical stimulation (Acu-LFES) attenuates denervation-induced muscle atrophy[J]. J Appl Physiol, 2016, 120(4): 426-436.
[10] Su Z, Robinson A, Hu L, et al.Acupuncture plus low-frequency electrical stimulation (Acu-LFES) attenuates diabetic myopathy by enhancing muscle regeneration[J]. PLoS One, 2015, 10(7): e0134511.
[11] 陈新旺,季传婷,虎力,等. 电针对衰老大鼠骨骼肌mTOR及磷酸化mTOR(ser2448)蛋白表达的影响[J]. 中华中医药杂志, 2017, 32(6): 2631-2633.
[12] 吴梦佳,唐成林,黄思琴,等. 电针对失神经大鼠腓肠肌中叉头蛋白转录因子3A、肌萎缩F-box蛋白及成肌分化抗原的影响[J/OL]. 针刺研究, 2018: 1-6 doi.org/10.13702/j.1000-0607.171015.
[13] Renault T, Chipuk J.Death upon a kiss: Mitochondrial Outer Membrane Composition and Organelle Communication Govern Sensitivity to BAK/BAX-Dependent Apoptosis[J]. Chem Biol, 2014, 21(1): 114-123.
[14] 高睿琦,唐成林,黄思琴,等. 电针对失坐骨神经大鼠腓肠肌细胞凋亡及相关蛋白的影响[J]. 针刺研究, 2017, 42(4): 302-307.
[15] 吴珍元,黄英如,冼华,等. 电针对失神经骨骼肌萎缩及纤维化的影响[J]. 中国康复医学杂志, 2016, 31(2): 177-182.
[16] 庄伊洢,黄晓卿,叶笑然,等.电针抗下肢肌萎缩的频率组合优化及其机制的初步研究[J]. 上海针灸杂志, 2017, 36(4): 473-477.
[17] Liu CC, Lin YC, Chen YH, et al.Cul3-KLHL20 ubiquitin ligase governs the turnover of ULK1 and VPS34 complexes to control autophagy termination[J]. Mol Cell, 2016, 61(1): 84-97.
[18] Schiaffino S, Dyar KA, Ciciliot S, et al.Mechanisms regulating skeletal muscle growth and atrophy[J]. FEBS J, 2013, 280(17): 4294-4314.
[19] 邵蕾,朱晓萍. 自噬-溶酶体途径与骨骼肌和膈肌萎缩的研究进展[J]. 复旦学报(医学版), 2017, 44(1): 99-104.
[20] Yang Y, Yang X, Dong Y, et al.Transcutaneous electrical acupoint stimulation alleviates adverse cardiac remodeling induced by overload training in rats[J]. J Appl Physiol, 2016, 120(11): 1269-1276.
[21] 尚画雨,付玉,夏志,等. 针刺对运动性骨骼肌损伤大鼠骨骼肌线粒体自噬的影响[J]. 中国病理生理杂志, 2017, 33(11): 2038-2046.
[22] 赵伟,刘延祥,王占魁,等. 自噬对电针治疗大鼠实验性坐骨神经损伤后神经再生的影响[J]. 天津中医药大学学报, 2014, 33(6): 351-354.
[23] 吴梦佳,唐成林,赵丹丹,等. 电针对失神经大鼠骨骼肌自噬相关基因表达的影响[J]. 中国康复理论与实践, 2018, 24(3): 260-265.
[24] Wang YX, Rudnicki MA.Satellite cells, the engines of muscle repair[J]. Nat Rev Mol Cell Biol, 2011, 13(2): 127-133.
[25] Tosic M, Allen A, Willmann D, et al.Lsd1 regulates skeletal muscle regeneration and directs the fate of satellite cells[J]. Nat Commun, 2018, 9(1): doi:10.1038/s41467-017-02740-5.
[26] Rodgers JT, Schroeder MD, Ma C, et al.HGFA Is an injury-regulated systemic factor that induces the transition of stem cells into GAlert[J]. Cell Rep, 2017, 19(3): 479-486.
[27] Hindi SM, Kumar A.TRAF6 regulates satellite stem cell self-renewal and function during regenerative myogenesis[J]. J Clin Invest, 2016, 126(1): 151-168.
[28] 熊慧,浦亚斌,马月辉,等. 骨骼肌卫星细胞自我更新的分子调节[J]. 生物医学工程学杂志, 2014, 31(5): 1168-1171.
[29] Sharma M, Mcfarlane C, Kambadur R, et al.Myostatin: expanding horizons[J]. IUBMB Life, 2015, 67(8): 589-600.
[30] 高睿琦,唐成林,曹净,等. 电针对失神经骨骼肌萎缩大鼠胰岛素样生长因子1、肌肉生长抑制素及肌卫星细胞增殖的影响[J]. 中国康复理论与实践, 2016, 22(11): 1259-1263.
[31] 高睿琦,唐成林,曹净,等. 电针对去神经支配骨骼肌萎缩大鼠PI3K/Akt/mTOR信号通路的影响[J]. 中国康复医学杂志, 2018, 33(1): 15-21.
[32] 郭秋平,文超越,王文龙,等. 肌纤维类型转化的分子信号通路及其营养调控进展[J]. 动物营养学报, 2017, 29(6): 1836-1842.
[33] 杨胜波,龙胜,易晓东,等. 注射性坐骨神经损伤后针刺对家兔腓肠肌内NADH-TR和胶原纤维的影响[J]. 遵义医学院学报, 2014, 37(1): 62-66.
[34] 张玮,苏利强,赵广高. 低频电刺激对坐骨神经损伤大鼠不同类型骨骼肌萎缩及内源性胰岛素样生长因子1表达的影响[J]. 中国组织工程研究与临床康复, 2011, 15(15): 2760-2763.
[35] 汪德生,张鹤,杜芳,等. 多穴位电针刺激对抗模拟失重大鼠比目鱼肌萎缩作用研究[J]. 航天医学与医学工程, 2017, 30(3): 180-184.
[36] Sigoillot SM, Bourgeois F, Karmouch J, et al.Neuromuscular junction immaturity and muscle atrophy are hallmarks of the ColQ-deficient mouse, a model of congenital myasthenic syndrome with acetylcholinesterase deficiency[J]. FASEB J, 2016, 30(6): 2382-2399.
[37] 李晓滨,曾园山,陈玉玲,等. 督脉电针与神经干细胞移植联合应用促进SCI横断大鼠前角运动神经元存活及减轻后肢肌萎缩的研究[J]. 中国康复医学杂志, 2006, 21(2): 104-107.
[38] Gorini A, Canosi U, Devecchi E, et al.ATPases enzyme activities during ageing in different types of somatic and synaptic plasma membranes from rat frontal cerebral cortex[J]. Prog Neuro-psychopharmacol Biol Psychiatry, 2002, 26(1): 81-90.
[39] 李曌嫱. 电针足三里对废用性肌肉萎缩大鼠Na^+-K^+-ATP酶活性的影响[J]. 内蒙古中医药, 2015, 34(1): 113-114.
[40] 潘华山,汶希,冯毅,等. 针刺对运动疲劳大鼠骨骼肌线粒体形态和游离Ca2+的影响[J]. 北京体育大学学报, 2011(10): 59-61.
[41] 庄伊洢,陈玄,叶笑然,等. 宽波电脉冲电针足三里防治下肢肌萎缩研究[J]. 上海针灸杂志, 2016, 35(6): 742-744.
[42] 袁婷,邵志成,欧婷,等. 电针治疗对大鼠坐骨神经损伤功能修复的作用[J]. 中国医学物理学杂志, 2017, 34(1): 105-108.
